In the therapy of functional ADA, cDNA is introduced into the lymphocytes by using vector a. Meloidegyne incognitia b. Agrobacterium Tumefaciens c. Salmonella typhimurium d. Retrovirus
Option D. The deficiency of the purine salvage enzyme adenosine deaminase is associated with severe B and T-cell dysfunction. In order to investigate the ADA molecule, gene therapy using E.coli, cDNA clones encoding ADA gene sequences are obtained.
